ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes.
Publication
, Journal Article
O'Sullivan-Murphy, B; Urano, F
Published in: Diabetes
April 2012
Duke Scholars
Published In
Diabetes
DOI
EISSN
1939-327X
Publication Date
April 2012
Volume
61
Issue
4
Start / End Page
780 / 781
Location
United States
Related Subject Headings
- Stress, Physiological
- Insulin-Secreting Cells
- Female
- Endoplasmic Reticulum
- Endocrinology & Metabolism
- Diabetes Mellitus, Type 1
- Animals
- 32 Biomedical and clinical sciences
- 11 Medical and Health Sciences
Citation
APA
Chicago
ICMJE
MLA
NLM
O’Sullivan-Murphy, B., & Urano, F. (2012). ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes. Diabetes, 61(4), 780–781. https://doi.org/10.2337/db12-0091
O’Sullivan-Murphy, Bryan, and Fumihiko Urano. “ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes.” Diabetes 61, no. 4 (April 2012): 780–81. https://doi.org/10.2337/db12-0091.
O’Sullivan-Murphy B, Urano F. ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes. Diabetes. 2012 Apr;61(4):780–1.
O’Sullivan-Murphy, Bryan, and Fumihiko Urano. “ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes.” Diabetes, vol. 61, no. 4, Apr. 2012, pp. 780–81. Pubmed, doi:10.2337/db12-0091.
O’Sullivan-Murphy B, Urano F. ER stress as a trigger for β-cell dysfunction and autoimmunity in type 1 diabetes. Diabetes. 2012 Apr;61(4):780–781.
Published In
Diabetes
DOI
EISSN
1939-327X
Publication Date
April 2012
Volume
61
Issue
4
Start / End Page
780 / 781
Location
United States
Related Subject Headings
- Stress, Physiological
- Insulin-Secreting Cells
- Female
- Endoplasmic Reticulum
- Endocrinology & Metabolism
- Diabetes Mellitus, Type 1
- Animals
- 32 Biomedical and clinical sciences
- 11 Medical and Health Sciences