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Identification of a protective proteomic signature and a potential therapeutic target in diabetic nephropathy.

Publication ,  Journal Article
Chun, N; Wyatt, CM; He, JC
Published in: Kidney Int
October 2017

In a recent paper in Nature Medicine, Qi et al. set out to identify protective pathways in patients with longstanding diabetes who do not develop diabetic nephropathy. Unbiased proteomic analysis of kidney tissue identified a unique protein signature in unaffected individuals. Studies in vitro and in murine models identified the highly upregulated glycolytic enzyme pyruvate kinase M2 as a potential therapeutic target to prevent or treat diabetic nephropathy.

Duke Scholars

Published In

Kidney Int

DOI

EISSN

1523-1755

Publication Date

October 2017

Volume

92

Issue

4

Start / End Page

780 / 781

Location

United States

Related Subject Headings

  • Urology & Nephrology
  • Pyruvate Kinase
  • Proteomics
  • Mitochondria
  • Mice
  • Kidney Glomerulus
  • Humans
  • Diabetic Nephropathies
  • Animals
  • 3202 Clinical sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Chun, N., Wyatt, C. M., & He, J. C. (2017). Identification of a protective proteomic signature and a potential therapeutic target in diabetic nephropathy. Kidney Int, 92(4), 780–781. https://doi.org/10.1016/j.kint.2017.08.002
Chun, Nicholas, Christina M. Wyatt, and John C. He. “Identification of a protective proteomic signature and a potential therapeutic target in diabetic nephropathy.Kidney Int 92, no. 4 (October 2017): 780–81. https://doi.org/10.1016/j.kint.2017.08.002.
Chun, Nicholas, et al. “Identification of a protective proteomic signature and a potential therapeutic target in diabetic nephropathy.Kidney Int, vol. 92, no. 4, Oct. 2017, pp. 780–81. Pubmed, doi:10.1016/j.kint.2017.08.002.
Journal cover image

Published In

Kidney Int

DOI

EISSN

1523-1755

Publication Date

October 2017

Volume

92

Issue

4

Start / End Page

780 / 781

Location

United States

Related Subject Headings

  • Urology & Nephrology
  • Pyruvate Kinase
  • Proteomics
  • Mitochondria
  • Mice
  • Kidney Glomerulus
  • Humans
  • Diabetic Nephropathies
  • Animals
  • 3202 Clinical sciences