Scholars@Duke

p19Arf is required for the cellular response to chronic DNA damage

Publication ,  Journal Article
Bieging-Rolett, KT; Johnson, TM; Brady, CA; Beaudry, VG; Pathak, N; Han, S; Attardi, LD
Published in: Oncogene
August 18, 2016
Published version (DOI) Publisher Link

Duke Scholars

Shuo Han
Author Shuo Han Biochemistry

Published In

Oncogene

DOI

10.1038/onc.2015.490

EISSN

1476-5594

ISSN

0950-9232

Publication Date

August 18, 2016

Volume

35

Issue

33

Start / End Page

4414 / 4421

Publisher

Springer Science and Business Media LLC

Related Subject Headings

  • Oncology & Carcinogenesis
  • 3211 Oncology and carcinogenesis
  • 3101 Biochemistry and cell biology
  • 1112 Oncology and Carcinogenesis
  • 1103 Clinical Sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Bieging-Rolett, K. T., Johnson, T. M., Brady, C. A., Beaudry, V. G., Pathak, N., Han, S., & Attardi, L. D. (2016). p19Arf is required for the cellular response to chronic DNA damage. Oncogene, 35(33), 4414–4421. https://doi.org/10.1038/onc.2015.490
Bieging-Rolett, K. T., T. M. Johnson, C. A. Brady, V. G. Beaudry, N. Pathak, S. Han, and L. D. Attardi. “p19Arf is required for the cellular response to chronic DNA damage.” Oncogene 35, no. 33 (August 18, 2016): 4414–21. https://doi.org/10.1038/onc.2015.490.
Bieging-Rolett KT, Johnson TM, Brady CA, Beaudry VG, Pathak N, Han S, et al. p19Arf is required for the cellular response to chronic DNA damage. Oncogene. 2016 Aug 18;35(33):4414–21.
Bieging-Rolett, K. T., et al. “p19Arf is required for the cellular response to chronic DNA damage.” Oncogene, vol. 35, no. 33, Springer Science and Business Media LLC, Aug. 2016, pp. 4414–21. Crossref, doi:10.1038/onc.2015.490.
Bieging-Rolett KT, Johnson TM, Brady CA, Beaudry VG, Pathak N, Han S, Attardi LD. p19Arf is required for the cellular response to chronic DNA damage. Oncogene. Springer Science and Business Media LLC; 2016 Aug 18;35(33):4414–4421.

Published In

Oncogene

DOI

10.1038/onc.2015.490

EISSN

1476-5594

ISSN

0950-9232

Publication Date

August 18, 2016

Volume

35

Issue

33

Start / End Page

4414 / 4421

Publisher

Springer Science and Business Media LLC

Related Subject Headings

  • Oncology & Carcinogenesis
  • 3211 Oncology and carcinogenesis
  • 3101 Biochemistry and cell biology
  • 1112 Oncology and Carcinogenesis
  • 1103 Clinical Sciences