Scholars@Duke
Journal cover image

Loss of UBE2N in keratinocytes leads to psoriasis-like skin inflammation and cutaneous dysbiosis characterized by corynebacterium outgrowth

Publication ,  Conference
Lee, M; Arnold, JW; Zhang, JY
Published in: JOURNAL OF INVESTIGATIVE DERMATOLOGY
2024

Duke Scholars

Jason Arnold
Author Jason Arnold Molecular Genetics and Microbiology

Published In

JOURNAL OF INVESTIGATIVE DERMATOLOGY

EISSN

1523-1747

ISSN

0022-202X

Publication Date

2024

Volume

144

Issue

8

Start / End Page

S84 / S84

Related Subject Headings

  • Dermatology & Venereal Diseases
  • 3202 Clinical sciences
  • 1112 Oncology and Carcinogenesis
  • 1103 Clinical Sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Lee, M., Arnold, J. W., & Zhang, J. Y. (2024). Loss of UBE2N in keratinocytes leads to psoriasis-like skin inflammation and cutaneous dysbiosis characterized by corynebacterium outgrowth. In JOURNAL OF INVESTIGATIVE DERMATOLOGY (Vol. 144, pp. S84–S84).
Lee, M., J. W. Arnold, and J. Y. Zhang. “Loss of UBE2N in keratinocytes leads to psoriasis-like skin inflammation and cutaneous dysbiosis characterized by corynebacterium outgrowth.” In JOURNAL OF INVESTIGATIVE DERMATOLOGY, 144:S84–S84, 2024.
Lee, M., et al. “Loss of UBE2N in keratinocytes leads to psoriasis-like skin inflammation and cutaneous dysbiosis characterized by corynebacterium outgrowth.” JOURNAL OF INVESTIGATIVE DERMATOLOGY, vol. 144, no. 8, 2024, pp. S84–S84.
Journal cover image

Published In

JOURNAL OF INVESTIGATIVE DERMATOLOGY

EISSN

1523-1747

ISSN

0022-202X

Publication Date

2024

Volume

144

Issue

8

Start / End Page

S84 / S84

Related Subject Headings

  • Dermatology & Venereal Diseases
  • 3202 Clinical sciences
  • 1112 Oncology and Carcinogenesis
  • 1103 Clinical Sciences