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319 The tumor-intrinsic NLRP3 inflammasome establishes a pulmonary metastatic niche via type II epithelial HSP70/TLR4 signaling and facilitates disease hyperprogression in response to immunotherapy

Publication ,  Conference
Theivanthiran, B; Liu, F; DeVito, N; Plebanek, M; Hanks, B
Published in: Regular and Young Investigator Award Abstracts
November 2021

Duke Scholars

Published In

Regular and Young Investigator Award Abstracts

DOI

Publication Date

November 2021

Start / End Page

A343 / A343

Publisher

BMJ Publishing Group Ltd

Conference Name

SITC 36th Anniversary Annual Meeting (SITC 2021) Abstracts

Related Subject Headings

  • 3211 Oncology and carcinogenesis
  • 3204 Immunology
 

Citation

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Theivanthiran, B., Liu, F., DeVito, N., Plebanek, M., & Hanks, B. (2021). 319 The tumor-intrinsic NLRP3 inflammasome establishes a pulmonary metastatic niche via type II epithelial HSP70/TLR4 signaling and facilitates disease hyperprogression in response to immunotherapy. In Regular and Young Investigator Award Abstracts (pp. A343–A343). BMJ Publishing Group Ltd. https://doi.org/10.1136/jitc-2021-sitc2021.319
Theivanthiran, Balamayooran, Fang Liu, Nicholas DeVito, Michael Plebanek, and Brent Hanks. “319 The tumor-intrinsic NLRP3 inflammasome establishes a pulmonary metastatic niche via type II epithelial HSP70/TLR4 signaling and facilitates disease hyperprogression in response to immunotherapy.” In Regular and Young Investigator Award Abstracts, A343–A343. BMJ Publishing Group Ltd, 2021. https://doi.org/10.1136/jitc-2021-sitc2021.319.
Theivanthiran B, Liu F, DeVito N, Plebanek M, Hanks B. 319 The tumor-intrinsic NLRP3 inflammasome establishes a pulmonary metastatic niche via type II epithelial HSP70/TLR4 signaling and facilitates disease hyperprogression in response to immunotherapy. In: Regular and Young Investigator Award Abstracts. BMJ Publishing Group Ltd; 2021. p. A343–A343.
Theivanthiran, Balamayooran, et al. “319 The tumor-intrinsic NLRP3 inflammasome establishes a pulmonary metastatic niche via type II epithelial HSP70/TLR4 signaling and facilitates disease hyperprogression in response to immunotherapy.” Regular and Young Investigator Award Abstracts, BMJ Publishing Group Ltd, 2021, pp. A343–A343. Crossref, doi:10.1136/jitc-2021-sitc2021.319.
Theivanthiran B, Liu F, DeVito N, Plebanek M, Hanks B. 319 The tumor-intrinsic NLRP3 inflammasome establishes a pulmonary metastatic niche via type II epithelial HSP70/TLR4 signaling and facilitates disease hyperprogression in response to immunotherapy. Regular and Young Investigator Award Abstracts. BMJ Publishing Group Ltd; 2021. p. A343–A343.

Published In

Regular and Young Investigator Award Abstracts

DOI

Publication Date

November 2021

Start / End Page

A343 / A343

Publisher

BMJ Publishing Group Ltd

Conference Name

SITC 36th Anniversary Annual Meeting (SITC 2021) Abstracts

Related Subject Headings

  • 3211 Oncology and carcinogenesis
  • 3204 Immunology