Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease.
Publication
, Journal Article
Abraham, DM; Marchuk, DA
Published in: Circ Res
March 14, 2014
Ischemia–reperfusion injury is strongly associated with increased oxidative stress, mitochondrial dysfunction, and cell death. These processes are diminished in an animal model of ischemia–reperfusion by the genetic loss or pharmacological inhibition of troponin I–interacting kinase.
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Published In
Circ Res
DOI
EISSN
1524-4571
Publication Date
March 14, 2014
Volume
114
Issue
6
Start / End Page
938 / 940
Location
United States
Related Subject Headings
- Ventricular Remodeling
- Protein Kinases
- Oxidative Stress
- Myocardial Ischemia
- MAP Kinase Kinase Kinases
- Humans
- Cardiovascular System & Hematology
- Animals
- 3202 Clinical sciences
- 3201 Cardiovascular medicine and haematology
Citation
APA
Chicago
ICMJE
MLA
NLM
Abraham, D. M., & Marchuk, D. A. (2014). Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease. Circ Res, 114(6), 938–940. https://doi.org/10.1161/CIRCRESAHA.113.303238
Abraham, Dennis M., and Douglas A. Marchuk. “Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease.” Circ Res 114, no. 6 (March 14, 2014): 938–40. https://doi.org/10.1161/CIRCRESAHA.113.303238.
Abraham DM, Marchuk DA. Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease. Circ Res. 2014 Mar 14;114(6):938–40.
Abraham, Dennis M., and Douglas A. Marchuk. “Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease.” Circ Res, vol. 114, no. 6, Mar. 2014, pp. 938–40. Pubmed, doi:10.1161/CIRCRESAHA.113.303238.
Abraham DM, Marchuk DA. Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease. Circ Res. 2014 Mar 14;114(6):938–940.
Published In
Circ Res
DOI
EISSN
1524-4571
Publication Date
March 14, 2014
Volume
114
Issue
6
Start / End Page
938 / 940
Location
United States
Related Subject Headings
- Ventricular Remodeling
- Protein Kinases
- Oxidative Stress
- Myocardial Ischemia
- MAP Kinase Kinase Kinases
- Humans
- Cardiovascular System & Hematology
- Animals
- 3202 Clinical sciences
- 3201 Cardiovascular medicine and haematology