Keeping autophagy in cheCK1.
Publication
, Journal Article
Cheong, JK; Virshup, DM
Published in: Mol Cell Oncol
May 2016
Mutant RAS-driven cancer cells cope with proliferative stress by increasing basal autophagy to maintain protein/organelle and energy homeostasis. We recently demonstrated that casein kinase 1 alpha (CK1α), a therapeutically tractable enzyme, is critical for fine-tuning the transcriptional regulation of mutant RAS-induced autophagy and the development of mutant RAS-driven cancers.
Duke Scholars
Published In
Mol Cell Oncol
DOI
ISSN
2372-3556
Publication Date
May 2016
Volume
3
Issue
3
Start / End Page
e1045117
Location
United States
Related Subject Headings
- 3211 Oncology and carcinogenesis
- 3205 Medical biochemistry and metabolomics
Citation
APA
Chicago
ICMJE
MLA
NLM
Cheong, J. K., & Virshup, D. M. (2016). Keeping autophagy in cheCK1. Mol Cell Oncol, 3(3), e1045117. https://doi.org/10.1080/23723556.2015.1045117
Cheong, Jit Kong, and David M. Virshup. “Keeping autophagy in cheCK1.” Mol Cell Oncol 3, no. 3 (May 2016): e1045117. https://doi.org/10.1080/23723556.2015.1045117.
Cheong JK, Virshup DM. Keeping autophagy in cheCK1. Mol Cell Oncol. 2016 May;3(3):e1045117.
Cheong, Jit Kong, and David M. Virshup. “Keeping autophagy in cheCK1.” Mol Cell Oncol, vol. 3, no. 3, May 2016, p. e1045117. Pubmed, doi:10.1080/23723556.2015.1045117.
Cheong JK, Virshup DM. Keeping autophagy in cheCK1. Mol Cell Oncol. 2016 May;3(3):e1045117.
Published In
Mol Cell Oncol
DOI
ISSN
2372-3556
Publication Date
May 2016
Volume
3
Issue
3
Start / End Page
e1045117
Location
United States
Related Subject Headings
- 3211 Oncology and carcinogenesis
- 3205 Medical biochemistry and metabolomics