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GLUTAMINE'S ATTENUATION OF NF-kB AND MORTALITY FOLLOWING SEPSIS ARE DEPENDENT ON HSP-70 EXPRESSION

Publication ,  Conference
Singleton, K; Tran, ZV; Wischmeyer, PE
Published in: Shock
June 2006

Duke Scholars

Published In

Shock

DOI

ISSN

1073-2322

Publication Date

June 2006

Volume

25

Issue

Supplement 1

Start / End Page

8 / 8

Publisher

Ovid Technologies (Wolters Kluwer Health)

Related Subject Headings

  • Emergency & Critical Care Medicine
  • 3202 Clinical sciences
  • 1103 Clinical Sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Singleton, K., Tran, Z. V., & Wischmeyer, P. E. (2006). GLUTAMINE'S ATTENUATION OF NF-kB AND MORTALITY FOLLOWING SEPSIS ARE DEPENDENT ON HSP-70 EXPRESSION. In Shock (Vol. 25, pp. 8–8). Ovid Technologies (Wolters Kluwer Health). https://doi.org/10.1097/00024382-200606001-00023
Singleton, K., Z. V. Tran, and P. E. Wischmeyer. “GLUTAMINE'S ATTENUATION OF NF-kB AND MORTALITY FOLLOWING SEPSIS ARE DEPENDENT ON HSP-70 EXPRESSION.” In Shock, 25:8–8. Ovid Technologies (Wolters Kluwer Health), 2006. https://doi.org/10.1097/00024382-200606001-00023.
Singleton K, Tran ZV, Wischmeyer PE. GLUTAMINE'S ATTENUATION OF NF-kB AND MORTALITY FOLLOWING SEPSIS ARE DEPENDENT ON HSP-70 EXPRESSION. In: Shock. Ovid Technologies (Wolters Kluwer Health); 2006. p. 8–8.
Singleton, K., et al. “GLUTAMINE'S ATTENUATION OF NF-kB AND MORTALITY FOLLOWING SEPSIS ARE DEPENDENT ON HSP-70 EXPRESSION.” Shock, vol. 25, no. Supplement 1, Ovid Technologies (Wolters Kluwer Health), 2006, pp. 8–8. Crossref, doi:10.1097/00024382-200606001-00023.
Singleton K, Tran ZV, Wischmeyer PE. GLUTAMINE'S ATTENUATION OF NF-kB AND MORTALITY FOLLOWING SEPSIS ARE DEPENDENT ON HSP-70 EXPRESSION. Shock. Ovid Technologies (Wolters Kluwer Health); 2006. p. 8–8.

Published In

Shock

DOI

ISSN

1073-2322

Publication Date

June 2006

Volume

25

Issue

Supplement 1

Start / End Page

8 / 8

Publisher

Ovid Technologies (Wolters Kluwer Health)

Related Subject Headings

  • Emergency & Critical Care Medicine
  • 3202 Clinical sciences
  • 1103 Clinical Sciences