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Biological model for assessing the role in glioma formation of genes differentially expressed in human gliomas

Publication ,  Conference
Rich, JN; Guo, CH; McLendon, RE; Bigner, DD; Wang, XF; Counter, CM
Published in: ANNALS OF NEUROLOGY
September 1, 2001

Duke Scholars

Published In

ANNALS OF NEUROLOGY

ISSN

0364-5134

Publication Date

September 1, 2001

Volume

50

Issue

3

Start / End Page

S31 / S31

Publisher

WILEY-LISS

Related Subject Headings

  • Neurology & Neurosurgery
  • 3209 Neurosciences
  • 3202 Clinical sciences
  • 1109 Neurosciences
  • 1103 Clinical Sciences
 

Citation

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ICMJE
MLA
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Rich, J. N., Guo, C. H., McLendon, R. E., Bigner, D. D., Wang, X. F., & Counter, C. M. (2001). Biological model for assessing the role in glioma formation of genes differentially expressed in human gliomas. In ANNALS OF NEUROLOGY (Vol. 50, pp. S31–S31). WILEY-LISS.
Rich, J. N., C. H. Guo, R. E. McLendon, D. D. Bigner, X. F. Wang, and C. M. Counter. “Biological model for assessing the role in glioma formation of genes differentially expressed in human gliomas.” In ANNALS OF NEUROLOGY, 50:S31–S31. WILEY-LISS, 2001.
Rich JN, Guo CH, McLendon RE, Bigner DD, Wang XF, Counter CM. Biological model for assessing the role in glioma formation of genes differentially expressed in human gliomas. In: ANNALS OF NEUROLOGY. WILEY-LISS; 2001. p. S31–S31.
Rich, J. N., et al. “Biological model for assessing the role in glioma formation of genes differentially expressed in human gliomas.” ANNALS OF NEUROLOGY, vol. 50, no. 3, WILEY-LISS, 2001, pp. S31–S31.
Rich JN, Guo CH, McLendon RE, Bigner DD, Wang XF, Counter CM. Biological model for assessing the role in glioma formation of genes differentially expressed in human gliomas. ANNALS OF NEUROLOGY. WILEY-LISS; 2001. p. S31–S31.
Journal cover image

Published In

ANNALS OF NEUROLOGY

ISSN

0364-5134

Publication Date

September 1, 2001

Volume

50

Issue

3

Start / End Page

S31 / S31

Publisher

WILEY-LISS

Related Subject Headings

  • Neurology & Neurosurgery
  • 3209 Neurosciences
  • 3202 Clinical sciences
  • 1109 Neurosciences
  • 1103 Clinical Sciences