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Cutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors

Publication ,  Journal Article
Escuin-Ordinas, H; Li, S; Xie, MW; Sun, L; Hugo, W; Huang, RR; Jiao, J; de-Faria, FM; Realegeno, S; Krystofinski, P; others
Published in: Nature communications
2016

Duke Scholars

Published In

Nature communications

Publication Date

2016

Volume

7

Publisher

Nature Research

Related Subject Headings

  • Wound Healing
  • Vemurafenib
  • Treatment Outcome
  • Sulfonamides
  • Skin Neoplasms
  • Skin
  • Pyrimidinones
  • Pyridones
  • Proto-Oncogene Proteins B-raf
  • Protein Kinase Inhibitors
 

Citation

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Escuin-Ordinas, H., Li, S., Xie, M. W., Sun, L., Hugo, W., Huang, R. R., … others. (2016). Cutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors. Nature Communications, 7.
Escuin-Ordinas, Helena, Shuoran Li, Michael W. Xie, Lu Sun, Willy Hugo, Rong Rong Huang, Jing Jiao, et al. “Cutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors.” Nature Communications 7 (2016).
Escuin-Ordinas H, Li S, Xie MW, Sun L, Hugo W, Huang RR, et al. Cutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors. Nature communications. 2016;7.
Escuin-Ordinas, Helena, et al. “Cutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors.” Nature Communications, vol. 7, Nature Research, 2016.
Escuin-Ordinas H, Li S, Xie MW, Sun L, Hugo W, Huang RR, Jiao J, de-Faria FM, Realegeno S, Krystofinski P, others. Cutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors. Nature communications. Nature Research; 2016;7.

Published In

Nature communications

Publication Date

2016

Volume

7

Publisher

Nature Research

Related Subject Headings

  • Wound Healing
  • Vemurafenib
  • Treatment Outcome
  • Sulfonamides
  • Skin Neoplasms
  • Skin
  • Pyrimidinones
  • Pyridones
  • Proto-Oncogene Proteins B-raf
  • Protein Kinase Inhibitors