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KEAP1 has a sweet spot: A new connection between intracellular glycosylation and redox stress signaling in cancer cells.

Journal articles  - Journal Article
Chen, P-H; Chi, J-T; Boyce, M
Published in: Mol Cell Oncol
2017

The KEAP1/NRF2 pathway is a master regulator of the redox stress response and is dysregulated in numerous human tumors. We discovered that NRF2 signaling is controlled by the site-specific glycosylation of KEAP1, revealing a potentially broad link among nutrient sensing, proteostasis and stress resistance in both normal and cancer cells.

Duke Scholars

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Published In

Mol Cell Oncol

DOI

ISSN

2372-3556

Publication Date

2017

Volume

4

Issue

6

Start / End Page

e1361501

Location

United States

Related Subject Headings

  • 3211 Oncology and carcinogenesis
  • 3205 Medical biochemistry and metabolomics
 

Citation

APA
Chicago
ICMJE
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Chen, P.-H., Chi, J.-T., & Boyce, M. (2017). KEAP1 has a sweet spot: A new connection between intracellular glycosylation and redox stress signaling in cancer cells. Mol Cell Oncol, 4(6), e1361501. https://doi.org/10.1080/23723556.2017.1361501
Chen, Po-Han, Jen-Tsan Chi, and Michael Boyce. “KEAP1 has a sweet spot: A new connection between intracellular glycosylation and redox stress signaling in cancer cells.Mol Cell Oncol 4, no. 6 (2017): e1361501. https://doi.org/10.1080/23723556.2017.1361501.
Chen, Po-Han, et al. “KEAP1 has a sweet spot: A new connection between intracellular glycosylation and redox stress signaling in cancer cells.Mol Cell Oncol, vol. 4, no. 6, 2017, p. e1361501. Pubmed, doi:10.1080/23723556.2017.1361501.

Published In

Mol Cell Oncol

DOI

ISSN

2372-3556

Publication Date

2017

Volume

4

Issue

6

Start / End Page

e1361501

Location

United States

Related Subject Headings

  • 3211 Oncology and carcinogenesis
  • 3205 Medical biochemistry and metabolomics