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Connecting cytokines and cellular signals in the nephron during CKD and hypertension.

Publication ,  Journal Article
Wen, Y; Crowley, SD
Published in: Kidney Int
April 2020

Chronic kidney disease features chronic inflammation and fibrosis, both of which contribute to and are exacerbated by arterial hypertension. The contribution of immune responses to renal sodium retention has received intense scrutiny. In this regard, the article by Furusho et al. details a mechanism wherein intrarenal TNFα augments salt-sensitive hypertension during CKD via activation of the WNK1-SPAKNCC phosphorylation cascade.

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Published In

Kidney Int

DOI

EISSN

1523-1755

Publication Date

April 2020

Volume

97

Issue

4

Start / End Page

651 / 653

Location

United States

Related Subject Headings

  • Urology & Nephrology
  • Tumor Necrosis Factor-alpha
  • Renal Insufficiency, Chronic
  • Phosphorylation
  • Nephrons
  • Kidney
  • Hypertension
  • Humans
  • Cytokines
  • 3202 Clinical sciences
 

Citation

APA
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ICMJE
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Wen, Y., & Crowley, S. D. (2020). Connecting cytokines and cellular signals in the nephron during CKD and hypertension. Kidney Int, 97(4), 651–653. https://doi.org/10.1016/j.kint.2020.01.005
Wen, Yi, and Steven D. Crowley. “Connecting cytokines and cellular signals in the nephron during CKD and hypertension.Kidney Int 97, no. 4 (April 2020): 651–53. https://doi.org/10.1016/j.kint.2020.01.005.
Wen, Yi, and Steven D. Crowley. “Connecting cytokines and cellular signals in the nephron during CKD and hypertension.Kidney Int, vol. 97, no. 4, Apr. 2020, pp. 651–53. Pubmed, doi:10.1016/j.kint.2020.01.005.
Wen Y, Crowley SD. Connecting cytokines and cellular signals in the nephron during CKD and hypertension. Kidney Int. 2020 Apr;97(4):651–653.
Journal cover image

Published In

Kidney Int

DOI

EISSN

1523-1755

Publication Date

April 2020

Volume

97

Issue

4

Start / End Page

651 / 653

Location

United States

Related Subject Headings

  • Urology & Nephrology
  • Tumor Necrosis Factor-alpha
  • Renal Insufficiency, Chronic
  • Phosphorylation
  • Nephrons
  • Kidney
  • Hypertension
  • Humans
  • Cytokines
  • 3202 Clinical sciences