
The Arf6 GAP centaurin α-1 is a neuronal actin-binding protein which also functions via GAP-independent activity to regulate the actin cytoskeleton
Publication
, Journal Article
Thacker, E; Kearns, B; Chapman, C; Hammond, J; Howell, A; Theibert, A
Published in: European Journal of Cell Biology
2004
Duke Scholars
Published In
European Journal of Cell Biology
DOI
ISSN
0171-9335
Publication Date
2004
Volume
83
Issue
10
Start / End Page
541 / 554
Publisher
Elsevier BV
Related Subject Headings
- Developmental Biology
- 3108 Plant biology
- 3101 Biochemistry and cell biology
- 0607 Plant Biology
- 0601 Biochemistry and Cell Biology
Citation
APA
Chicago
ICMJE
MLA
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Thacker, E., Kearns, B., Chapman, C., Hammond, J., Howell, A., & Theibert, A. (2004). The Arf6 GAP centaurin α-1 is a neuronal actin-binding protein which also functions via GAP-independent activity to regulate the actin cytoskeleton. European Journal of Cell Biology, 83(10), 541–554. https://doi.org/10.1078/0171-9335-00416
Thacker, Erin, Brian Kearns, Carlene Chapman, Jennifer Hammond, Audrey Howell, and Anne Theibert. “The Arf6 GAP centaurin α-1 is a neuronal actin-binding protein which also functions via GAP-independent activity to regulate the actin cytoskeleton.” European Journal of Cell Biology 83, no. 10 (2004): 541–54. https://doi.org/10.1078/0171-9335-00416.
Thacker E, Kearns B, Chapman C, Hammond J, Howell A, Theibert A. The Arf6 GAP centaurin α-1 is a neuronal actin-binding protein which also functions via GAP-independent activity to regulate the actin cytoskeleton. European Journal of Cell Biology. 2004;83(10):541–54.
Thacker, Erin, et al. “The Arf6 GAP centaurin α-1 is a neuronal actin-binding protein which also functions via GAP-independent activity to regulate the actin cytoskeleton.” European Journal of Cell Biology, vol. 83, no. 10, Elsevier BV, 2004, pp. 541–54. Crossref, doi:10.1078/0171-9335-00416.
Thacker E, Kearns B, Chapman C, Hammond J, Howell A, Theibert A. The Arf6 GAP centaurin α-1 is a neuronal actin-binding protein which also functions via GAP-independent activity to regulate the actin cytoskeleton. European Journal of Cell Biology. Elsevier BV; 2004;83(10):541–554.

Published In
European Journal of Cell Biology
DOI
ISSN
0171-9335
Publication Date
2004
Volume
83
Issue
10
Start / End Page
541 / 554
Publisher
Elsevier BV
Related Subject Headings
- Developmental Biology
- 3108 Plant biology
- 3101 Biochemistry and cell biology
- 0607 Plant Biology
- 0601 Biochemistry and Cell Biology