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Tobacco carcinogen-induced cellular transformation increases Akt activation in vitro and in vivo.

Publication ,  Conference
West, KA; Linnoila, IR; Brognard, J; Belinsky, S; Harris, C; Dennis, PA
Published in: Chest
May 2004

Duke Scholars

Published In

Chest

DOI

ISSN

0012-3692

Publication Date

May 2004

Volume

125

Issue

5 Suppl

Start / End Page

101S / 102S

Location

United States

Related Subject Headings

  • Respiratory System
  • Proto-Oncogene Proteins c-akt
  • Proto-Oncogene Proteins
  • Protein Serine-Threonine Kinases
  • Phosphorylation
  • Nitrosamines
  • Mice, Inbred A
  • Mice
  • Lung Neoplasms
  • Humans
 

Citation

APA
Chicago
ICMJE
MLA
NLM
West, K. A., Linnoila, I. R., Brognard, J., Belinsky, S., Harris, C., & Dennis, P. A. (2004). Tobacco carcinogen-induced cellular transformation increases Akt activation in vitro and in vivo. In Chest (Vol. 125, pp. 101S-102S). United States. https://doi.org/10.1378/chest.125.5_suppl.101s
West, Kip A., Ilona R. Linnoila, John Brognard, Steven Belinsky, Curtis Harris, and Phillip A. Dennis. “Tobacco carcinogen-induced cellular transformation increases Akt activation in vitro and in vivo.” In Chest, 125:101S-102S, 2004. https://doi.org/10.1378/chest.125.5_suppl.101s.
West KA, Linnoila IR, Brognard J, Belinsky S, Harris C, Dennis PA. Tobacco carcinogen-induced cellular transformation increases Akt activation in vitro and in vivo. In: Chest. 2004. p. 101S-102S.
West, Kip A., et al. “Tobacco carcinogen-induced cellular transformation increases Akt activation in vitro and in vivo.Chest, vol. 125, no. 5 Suppl, 2004, pp. 101S-102S. Pubmed, doi:10.1378/chest.125.5_suppl.101s.
West KA, Linnoila IR, Brognard J, Belinsky S, Harris C, Dennis PA. Tobacco carcinogen-induced cellular transformation increases Akt activation in vitro and in vivo. Chest. 2004. p. 101S-102S.
Journal cover image

Published In

Chest

DOI

ISSN

0012-3692

Publication Date

May 2004

Volume

125

Issue

5 Suppl

Start / End Page

101S / 102S

Location

United States

Related Subject Headings

  • Respiratory System
  • Proto-Oncogene Proteins c-akt
  • Proto-Oncogene Proteins
  • Protein Serine-Threonine Kinases
  • Phosphorylation
  • Nitrosamines
  • Mice, Inbred A
  • Mice
  • Lung Neoplasms
  • Humans