Mitochondrial Calcium Entry Induces Mitochondrial Reactive Oxygen Species Production by ATP Synthase-uncoupled, But Proton Leak-coupled Mechanism - A New System of How Atherogenic Stimuli Activate Aortic Endothelial Cells without Compromising Endothelial Cell Survival
Publication
, Conference
Li, X; Fu, H; Fang, P; Koch, WJ; Wang, H; Yang, X-F
Published in: FASEB JOURNAL
2016
Duke Scholars
Published In
FASEB JOURNAL
EISSN
1530-6860
ISSN
0892-6638
Publication Date
2016
Volume
30
Related Subject Headings
- Biochemistry & Molecular Biology
- 3208 Medical physiology
- 3101 Biochemistry and cell biology
- 1116 Medical Physiology
- 0606 Physiology
- 0601 Biochemistry and Cell Biology
Citation
APA
Chicago
ICMJE
MLA
NLM
Li, X., Fu, H., Fang, P., Koch, W. J., Wang, H., & Yang, X.-F. (2016). Mitochondrial Calcium Entry Induces Mitochondrial Reactive Oxygen Species Production by ATP Synthase-uncoupled, But Proton Leak-coupled Mechanism - A New System of How Atherogenic Stimuli Activate Aortic Endothelial Cells without Compromising Endothelial Cell Survival. In FASEB JOURNAL (Vol. 30).
Li, Xinyuan, Hangfei Fu, Pu Fang, Walter J. Koch, Hong Wang, and Xiao-Feng Yang. “Mitochondrial Calcium Entry Induces Mitochondrial Reactive Oxygen Species Production by ATP Synthase-uncoupled, But Proton Leak-coupled Mechanism - A New System of How Atherogenic Stimuli Activate Aortic Endothelial Cells without Compromising Endothelial Cell Survival.” In FASEB JOURNAL, Vol. 30, 2016.
Li X, Fu H, Fang P, Koch WJ, Wang H, Yang X-F. Mitochondrial Calcium Entry Induces Mitochondrial Reactive Oxygen Species Production by ATP Synthase-uncoupled, But Proton Leak-coupled Mechanism - A New System of How Atherogenic Stimuli Activate Aortic Endothelial Cells without Compromising Endothelial Cell Survival. In: FASEB JOURNAL. 2016.
Li, Xinyuan, et al. “Mitochondrial Calcium Entry Induces Mitochondrial Reactive Oxygen Species Production by ATP Synthase-uncoupled, But Proton Leak-coupled Mechanism - A New System of How Atherogenic Stimuli Activate Aortic Endothelial Cells without Compromising Endothelial Cell Survival.” FASEB JOURNAL, vol. 30, 2016.
Li X, Fu H, Fang P, Koch WJ, Wang H, Yang X-F. Mitochondrial Calcium Entry Induces Mitochondrial Reactive Oxygen Species Production by ATP Synthase-uncoupled, But Proton Leak-coupled Mechanism - A New System of How Atherogenic Stimuli Activate Aortic Endothelial Cells without Compromising Endothelial Cell Survival. FASEB JOURNAL. 2016.
Published In
FASEB JOURNAL
EISSN
1530-6860
ISSN
0892-6638
Publication Date
2016
Volume
30
Related Subject Headings
- Biochemistry & Molecular Biology
- 3208 Medical physiology
- 3101 Biochemistry and cell biology
- 1116 Medical Physiology
- 0606 Physiology
- 0601 Biochemistry and Cell Biology