Genetic deletion of beta-arrestin-1 improves function of the infarcted heart by reducing cardiotoxic neurohormonal overstimulation
Publication
, Conference
Lymperopoulos, A; Rengo, G; Gao, E; Zincarelli, C; Koch, WJ
Published in: CARDIOVASCULAR RESEARCH
2010
Duke Scholars
Published In
CARDIOVASCULAR RESEARCH
ISSN
0008-6363
Publication Date
2010
Volume
87
Start / End Page
S92 / S93
Related Subject Headings
- Cardiovascular System & Hematology
- 3201 Cardiovascular medicine and haematology
- 1102 Cardiorespiratory Medicine and Haematology
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APA
Chicago
ICMJE
MLA
NLM
Lymperopoulos, A., Rengo, G., Gao, E., Zincarelli, C., & Koch, W. J. (2010). Genetic deletion of beta-arrestin-1 improves function of the infarcted heart by reducing cardiotoxic neurohormonal overstimulation. In CARDIOVASCULAR RESEARCH (Vol. 87, pp. S92–S93).
Lymperopoulos, A., G. Rengo, E. Gao, C. Zincarelli, and W. J. Koch. “Genetic deletion of beta-arrestin-1 improves function of the infarcted heart by reducing cardiotoxic neurohormonal overstimulation.” In CARDIOVASCULAR RESEARCH, 87:S92–93, 2010.
Lymperopoulos A, Rengo G, Gao E, Zincarelli C, Koch WJ. Genetic deletion of beta-arrestin-1 improves function of the infarcted heart by reducing cardiotoxic neurohormonal overstimulation. In: CARDIOVASCULAR RESEARCH. 2010. p. S92–3.
Lymperopoulos, A., et al. “Genetic deletion of beta-arrestin-1 improves function of the infarcted heart by reducing cardiotoxic neurohormonal overstimulation.” CARDIOVASCULAR RESEARCH, vol. 87, 2010, pp. S92–93.
Lymperopoulos A, Rengo G, Gao E, Zincarelli C, Koch WJ. Genetic deletion of beta-arrestin-1 improves function of the infarcted heart by reducing cardiotoxic neurohormonal overstimulation. CARDIOVASCULAR RESEARCH. 2010. p. S92–S93.
Published In
CARDIOVASCULAR RESEARCH
ISSN
0008-6363
Publication Date
2010
Volume
87
Start / End Page
S92 / S93
Related Subject Headings
- Cardiovascular System & Hematology
- 3201 Cardiovascular medicine and haematology
- 1102 Cardiorespiratory Medicine and Haematology