Cardiac S100a1 protein levels determine left ventricular performance and propensity towards heart failure after myocardial infarction in mice
Publication
, Conference
Most, P; Gao, E; Funakoshi, H; Voelkers, M; Heierhorst, J; Eckhart, A; Pleger, ST; Feldman, AM; Katus, HA; Koch, WJ
Published in: CIRCULATION
2005
Duke Scholars
Published In
CIRCULATION
ISSN
0009-7322
Publication Date
2005
Volume
112
Issue
17
Start / End Page
U240 / U241
Related Subject Headings
- Cardiovascular System & Hematology
- 4207 Sports science and exercise
- 3202 Clinical sciences
- 3201 Cardiovascular medicine and haematology
- 1117 Public Health and Health Services
- 1103 Clinical Sciences
- 1102 Cardiorespiratory Medicine and Haematology
Citation
APA
Chicago
ICMJE
MLA
NLM
Most, P., Gao, E., Funakoshi, H., Voelkers, M., Heierhorst, J., Eckhart, A., … Koch, W. J. (2005). Cardiac S100a1 protein levels determine left ventricular performance and propensity towards heart failure after myocardial infarction in mice. In CIRCULATION (Vol. 112, pp. U240–U241).
Most, P., E. Gao, H. Funakoshi, M. Voelkers, J. Heierhorst, A. Eckhart, S. T. Pleger, A. M. Feldman, H. A. Katus, and W. J. Koch. “Cardiac S100a1 protein levels determine left ventricular performance and propensity towards heart failure after myocardial infarction in mice.” In CIRCULATION, 112:U240–41, 2005.
Most P, Gao E, Funakoshi H, Voelkers M, Heierhorst J, Eckhart A, et al. Cardiac S100a1 protein levels determine left ventricular performance and propensity towards heart failure after myocardial infarction in mice. In: CIRCULATION. 2005. p. U240–1.
Most, P., et al. “Cardiac S100a1 protein levels determine left ventricular performance and propensity towards heart failure after myocardial infarction in mice.” CIRCULATION, vol. 112, no. 17, 2005, pp. U240–41.
Most P, Gao E, Funakoshi H, Voelkers M, Heierhorst J, Eckhart A, Pleger ST, Feldman AM, Katus HA, Koch WJ. Cardiac S100a1 protein levels determine left ventricular performance and propensity towards heart failure after myocardial infarction in mice. CIRCULATION. 2005. p. U240–U241.
Published In
CIRCULATION
ISSN
0009-7322
Publication Date
2005
Volume
112
Issue
17
Start / End Page
U240 / U241
Related Subject Headings
- Cardiovascular System & Hematology
- 4207 Sports science and exercise
- 3202 Clinical sciences
- 3201 Cardiovascular medicine and haematology
- 1117 Public Health and Health Services
- 1103 Clinical Sciences
- 1102 Cardiorespiratory Medicine and Haematology