Skip to main content
Journal cover image

Expression of the 71 kDa dystrophin isoform (Dp71) evaluated by gene targeting.

Publication ,  Journal Article
Lumeng, CN; Hauser, M; Brown, V; Chamberlain, JS
Published in: Brain Res
May 29, 1999

To investigate the function of the major non-muscle dystrophin isoform, Dp71, we substituted a beta-galactosidase (betagal) reporter gene for Dp71 by homologous recombination in embryonic stem cells. Staining for betagal activity in chimeric mice revealed Dp71 promoter activity in glial cells in the CNS, in neurons of the inner nuclear and inner plexiform layers of the retina, and in the kidney tubules and collecting ducts. Our observations demonstrate that Dp71 is widely expressed in the adult CNS (retina, cerebellum, cerebral cortex, ependyma, and choroid) as well as the adult kidney epithelium and suggest a broad function for Dp71 in differentiated tissues.

Duke Scholars

Published In

Brain Res

DOI

ISSN

0006-8993

Publication Date

May 29, 1999

Volume

830

Issue

1

Start / End Page

174 / 178

Location

Netherlands

Related Subject Headings

  • beta-Galactosidase
  • Protein Isoforms
  • Neurology & Neurosurgery
  • Molecular Weight
  • Mice, Inbred C57BL
  • Mice
  • In Situ Hybridization
  • Genes, Reporter
  • Gene Targeting
  • Gene Expression Regulation
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Lumeng, C. N., Hauser, M., Brown, V., & Chamberlain, J. S. (1999). Expression of the 71 kDa dystrophin isoform (Dp71) evaluated by gene targeting. Brain Res, 830(1), 174–178. https://doi.org/10.1016/s0006-8993(99)01201-9
Lumeng, C. N., M. Hauser, V. Brown, and J. S. Chamberlain. “Expression of the 71 kDa dystrophin isoform (Dp71) evaluated by gene targeting.Brain Res 830, no. 1 (May 29, 1999): 174–78. https://doi.org/10.1016/s0006-8993(99)01201-9.
Lumeng CN, Hauser M, Brown V, Chamberlain JS. Expression of the 71 kDa dystrophin isoform (Dp71) evaluated by gene targeting. Brain Res. 1999 May 29;830(1):174–8.
Lumeng, C. N., et al. “Expression of the 71 kDa dystrophin isoform (Dp71) evaluated by gene targeting.Brain Res, vol. 830, no. 1, May 1999, pp. 174–78. Pubmed, doi:10.1016/s0006-8993(99)01201-9.
Lumeng CN, Hauser M, Brown V, Chamberlain JS. Expression of the 71 kDa dystrophin isoform (Dp71) evaluated by gene targeting. Brain Res. 1999 May 29;830(1):174–178.
Journal cover image

Published In

Brain Res

DOI

ISSN

0006-8993

Publication Date

May 29, 1999

Volume

830

Issue

1

Start / End Page

174 / 178

Location

Netherlands

Related Subject Headings

  • beta-Galactosidase
  • Protein Isoforms
  • Neurology & Neurosurgery
  • Molecular Weight
  • Mice, Inbred C57BL
  • Mice
  • In Situ Hybridization
  • Genes, Reporter
  • Gene Targeting
  • Gene Expression Regulation