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The role of G-protein-coupled receptors in heart failure

Publication ,  Journal Article
Lemaire, A; Rockman, HA
Published in: Drug Discovery Today Disease Mechanisms
January 1, 2004

Central to the development of congestive heart failure is the role by G-protein-coupled receptors (GPCRs). The sympathetic nervous system stimulates GPCRs during periods of stress. Novel therapeutic approaches to heart failure are focused on key enzymes that regulate the transduction pathways. The purpose of this review is to document the mechanism by which an alteration in the GPCR transduction processes contributes to the pathophysiology of heart failure. © 2004 Elsevier Ltd. All rights reserved.

Duke Scholars

Published In

Drug Discovery Today Disease Mechanisms

DOI

ISSN

1740-6765

Publication Date

January 1, 2004

Volume

1

Issue

1

Start / End Page

37 / 43

Related Subject Headings

  • Immunology
 

Citation

APA
Chicago
ICMJE
MLA
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Lemaire, A., & Rockman, H. A. (2004). The role of G-protein-coupled receptors in heart failure. Drug Discovery Today Disease Mechanisms, 1(1), 37–43. https://doi.org/10.1016/j.ddmec.2004.08.013
Lemaire, A., and H. A. Rockman. “The role of G-protein-coupled receptors in heart failure.” Drug Discovery Today Disease Mechanisms 1, no. 1 (January 1, 2004): 37–43. https://doi.org/10.1016/j.ddmec.2004.08.013.
Lemaire A, Rockman HA. The role of G-protein-coupled receptors in heart failure. Drug Discovery Today Disease Mechanisms. 2004 Jan 1;1(1):37–43.
Lemaire, A., and H. A. Rockman. “The role of G-protein-coupled receptors in heart failure.” Drug Discovery Today Disease Mechanisms, vol. 1, no. 1, Jan. 2004, pp. 37–43. Scopus, doi:10.1016/j.ddmec.2004.08.013.
Lemaire A, Rockman HA. The role of G-protein-coupled receptors in heart failure. Drug Discovery Today Disease Mechanisms. 2004 Jan 1;1(1):37–43.
Journal cover image

Published In

Drug Discovery Today Disease Mechanisms

DOI

ISSN

1740-6765

Publication Date

January 1, 2004

Volume

1

Issue

1

Start / End Page

37 / 43

Related Subject Headings

  • Immunology